Fiona Hatch
About
Biography
- Post Doctorate Research Fellow, University of Surrey 2013
- PhD Cardiac Physiology, University of Hull 2009-2012
- BSc Biological Sciences, University of Bristol 2006-2009
Research interests
Fiona is a cardiac physiologist focused on pathologies such as atrial fibrillation. Her work is funded by the Heart and Stroke Trust Endevour (HASTE) grant to investigate atrial fibrillation. This has developed into determining the role of connexin modification in conduction and the implications in the onset of arrhythmias.
Professional Affliations
The Physiological Society - Affliate Representative
Publications
Additional publications
<p>Hatch F, Lancaster MK & Jones SA. <a href="http://www.physoc.org/proceedings/abstract/Proc%2037th%20IUPSPCB021">Do… the crista terminalis possess a key role in the elevated risk of arrhythmogenic episodes in the elderly population?</a> Proc 37th IUPS (2013) PCB021</p><p>Hatch F, Lancaster MK & Jones SA. <a href="http://www.physoc.org/proceedings/abstract/Proc%2037th%20IUPSPCB022">Ag… further increases the diversification between the atria in the expression of cellular calcium regulators: a substrate for increase arrhythmia risk?</a> Proc 37th IUPS (2013) PCB022</p><p>Hatch F, Lancaster MK & Jones SA. <a href="http://www.physoc.org/proceedings/abstract/Proc%20Physiol%20Soc%2028C04… alterations in intracellular calcium handling within the sinoatrial node.</a> Proc Physiol Soc 2012:28;C04&PC04</p><p>Hatch F, Lancaster MK & Jones SA. <a href="http://www.physoc.org/proceedings/abstract/Proc%20Physiol%20Soc%2028PC3… SR function and adrenergic response co-associate in the ageing sinoatrial node.</a> Proc Physiol Soc 2012:28;PC38</p><p>Hatch F, Lancaster MK & Jones SA. <a href="http://heart.bmj.com/content/98/Suppl_3/A5.4">Changes in the role of the SR in SAN function across the lifespan may be responsible for changing pacemaker stability and response.</a> Heart 2012;98(3);A17</p><p>Hatch F, Lancaster MK & Jones SA. <a href="http://www.ncbi.nlm.nih.gov/pubmed/21878050">Aging is a primary risk factor for cardiac arrhythmias: disruption of intracellular Ca2+ regulation as a key suspect.</a> Expert Rev Cardiovasc Ther 9(8), 1059-1067 (2011).</p>