Mike Cathcart studied at School of Veterinary Medicine, University of Glasgow, graduating with an intercalated BSc (Microbiology) in 2000 and bachelor of veterinary medicine & surgery in 2002. Working in private practice for 6 years, Mike developed a keen interest in all aspects of equine practice, but specifically in equine medicine, which led to him returning to Glasgow Vet School in 2008 to complete a 4-year clinical scholarship in equine internal medicine, supported by the Horserace Betting Levy Board.
Mike obtained the RCVS certificate in equine internal medicine in 2010, became a diplomat of the European College of Equine Internal Medicine in 2013, and graduated with a Masters in Veterinary Medicine (MVM) from Glasgow University in 2016.
Following a 3-year position at the University of Adelaide School of Animal & Veterinary Sciences, where he directed the clinical component of the undergraduate equine curriculum, Mike returned to the UK in March 2016 when he joined the School of Veterinary Medicine as Senior Teaching Fellow in Equine Studies.
Mike has a broad range of research interests within the field of equine internal medicine. He completed an HBLB-funded Masters research project, developing non-invasive methods of detecting lower airway inflammation in Thoroughbred racehorses, and has subsequently completed research in equine metabolic and endocrinopathic disease. Mike is active in supervising undergraduate student research projects, and in engaging private veterinary practitioners in clinical research.
As Senior Teaching Fellow in Equine Studies, Mike will engage with and contribute to all levels of the curriculum, ensuring there is continuity and appropriate integration of equine clinical teaching throughout the course. His specific focus is on the clinical practice modules in year 3 and the equine clinical module in year 4. Mike will also work closely with the private partner clinics to facilitate the equine teaching in the final year.
VMS3007: Clinical Module 4
- Equine species lead in veterinary clinical practice
- Section lead within Department of Veterinary Clinical Studies
- Chair of the assessment working group
- Royal College of Veterinary Surgeons
- European College of Equine Internal Medicine
- British Equine Veterinary Association
exhaled breath condensate (EBC) have been established as methods
for the diagnosis and monitoring of lung inflammatory diseases
in human medicine. The application of non-invasive
diagnostic techniques in the investigation of lower airway inflammation
in horses holds much appeal. The aims of this study were
to (i) investigate whether both EB and EBC could be obtained
from Thoroughbred racehorses in the field setting, and (ii) investigate
the effects of exercise per se on the concentrations of
exhaled nitric oxide (NO) and carbon monoxide (CO) and EBC
common condition in horses that is associated with insulin resistance
(IR) and high morbidity and mortality in affected cases
that develop laminitis. Metformin is used in humans with IR and
may be of clinical benefit in horses with EMS. The aim of this
study was to determine whether the administration of metformin,
in conjunction with dietary restriction in horses with EMS,
results in increased insulin sensitivity (IS).
eNO was detected in 19/84 EB samples. eCO was measured in 39/42 EB samples pre-exercise (median 1.3 ppm) and concentrations decreased significantly post-exercise (median 0.8 ppm, P
reported in equids as an incidental post mortem finding,
infrequently associated with clinical signs of oesophageal
dysfunction or additional oesophageal abnormalities.
Clinicopathological and post mortem findings are presented
from a 15-year-old gelding that developed a spontaneous
perforation of the distal, intrathoracic oesophagus, in
association with IMHO. The majority of oesophageal
perforations documented in the equine literature have been
related to external or iatrogenic trauma resulting in rupture of
the cervical portion of the oesophagus. In this case, the early
presenting features included pyrexia and inappetance, and
progressed over a 4 day period to include signs of
tachypnoea, tachycardia and endotoxaemia due to the
development of a fatal septic pleuritis and pleural effusion.
Oesophageal obstruction was not a presenting feature.
Diagnostic investigation in cases of unexplained septic pleural
effusion should include oesophageal endoscopic evaluation
to rule out perforation of the intrathoracic oesophagus. IMHO
may represent a clinically significant, primary disease entity
with the potential to lead to oesophageal perforation.
Sampler design was found to significantly affect pH. Samples collected as a liquid had a significantly higher pH than samples frozen during collection (P
A comparison of the approaches used in veterinary research by different groups may aid in the identification of potentially reliable and repeatable biomarkers suitable for further investigation. To date, changes in acid?base status and increased concentrations of inflammatory mediators have been the main findings in studies of pulmonary disease states in animals. Whilst these biomarkers are unlikely to represent specific and sensitive diagnostic parameters, they do have potential application in monitoring disease progression and treatment response.